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Cardioprotective effects documented in ischaemia-reperfusion injury models, with mitochondrial membrane integrity preserved and ROS-related damage attenuated under oxidative stress conditions
Reduced mitochondrial ROS production and improved ATP synthesis efficiency observed in preclinical research, without inhibition of the electron transport chain — a selective protective mechanism
Improvements in exercise capacity, skeletal muscle function, and mitochondrial cristae architecture noted in sarcopenia and age-related decline research models, consistent with cardiolipin stabilisation activity
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